| 1. Possible signs of posterior column damage include: | |
| A. Negative rombergism |
F
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| B. Diminished tendon reflexes |
T
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| C. Hypertonicity |
F
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| D. Loss of vibration sense |
T
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| E. Loss of proprioception |
T
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| 2. Features of Alzheimer’s disease may include: | |
| A. Glial proliferation |
T
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| B. Cystic necrosis and gliosis |
F
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| C. Granulovacuolar degeneration |
T
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| D. Multiple micro-infarcts |
F
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| E. Arteriosclerosis |
F
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| 3. Features of upper motor neuron lesions could include: | |
| A. Clonus |
T
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| B. Cogwheel rigidity |
F
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| C. Flexor plantar response |
F
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| D. Preservation of muscle bulk |
T
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| E. Increased tendon reflexes |
T
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| 4. Features of Pick’s disease might include: | |
| A. Frontal and parietal lobes mainly affected |
F
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| B. ‘knife-blade’ atrophy |
T
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| C. ‘balloon cells’ |
T
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| D. Absence of fibrous gliosis |
F
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| E. Pick’s cells |
T
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| 5. Activation techniques in electroencephalography include: | |
| A. Dehydration |
F
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| B. Sodium valproate |
F
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| C. Alcohol |
T
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| D. Hypoventilation |
F
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| E. Sleep |
T
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| 6. Features of Creutzfeldt-Jacob disease include: | |
| A. Neuronal degeneration |
T
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| B. Atrophy of caudate and putamen especially |
F
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| C. Spongeiform changes |
T
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| D. Does not affect the whole CNS |
F
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| E. Glial proliferation |
T
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| 7. Possible features of lower motor neuron disease include: | |
| A. Atonic muscles |
T
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| B. Absent reflexes |
T
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| C. Loss of 100 % of muscle bulk |
F
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| D. Clasp-knife rigidity |
F
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| E. Tardive dyskinesia |
F
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| 8. Features of multi-infarct dementia include: | |
| A. Senile plaques |
F
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| B. Neurofibrillary tangles |
F
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| C. Hypertension |
T
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| D. Cerebral ischaemia |
T
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| E. Cerebral infarction |
T
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| 9. Functions of the non-dominant cerebral hemisphere may include: | |
| A. Holistic |
T
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| B. Ideational |
F
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| C. Pictorial |
T
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| D. Geometric |
T
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| E. Non-linear |
T
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| 10. Memory defects occur with lesions in: | |
| A. Medial-dorsal thalamic nucleus |
T
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| B. Wernicke’s area |
F
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| C. Walls of third ventricle |
T
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| D. Broca’s area |
F
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| E. Parietal cortex |
F
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| 11. The limbic system includes: | |
| A. Parahippocampal gyrus |
T
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| B. Hypothalamus |
T
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| C. Corpus callosum |
F
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| D. Anterior nucleus of thalamus |
T
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| E. Subcallosal gyrus |
T
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| 12. Diplopia occurs in: | |
| A. Neuropathy of oculomotor nerve |
T
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| B. Parkinson’s disease |
F
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| C. Neuropathy of facial nerve |
F
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| D. Huntington’s disease |
F
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| E. Diabetes insipidus |
F
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| 13. Associations of benign intracranial hypertension might include: | |
| A. Chlortetracycline administration |
T
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| B. Myxoedema |
F
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| C. Polycythaemia |
T
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| D. Oral contraceptives |
T
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| E. Hypoparathyroidism |
T
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| 14. Structures involved in the accommodation reflex include: | |
| A. Pretectal nucleus |
F
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| B. Edinger-Westphal nucleus (bilaterally) |
F
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| C. Lateral geniculate body |
T
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| D. Inferior colliculus |
F
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| E. Oculomotor nuclei of midbrain |
T
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| 15. Features of posterior inferior cerebellar artery occlusion might include: | |
| A. Contralateral Horner’s syndrome |
F
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| B. Ipsilateral analgesia (limbs) |
F
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| C. Contralateral ataxia |
F
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| D. Dissociated analgesia |
T
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| E. Ipsilateral analgesia (facial) |
T
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| 16. Nystagmus occurs in: | |
| A. Brain stem lesions |
T
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| B. Labyrinthine disease |
T
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| C. Cerebellar lesions |
T
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| D. Healthy subjects |
T
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| E. Rotational stimulation |
T
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| 17. Features of basilar artery occlusion could include: | |
| A. Monoplegia |
T
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| B. Contralateral cerebellar signs |
F
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| C. Quadriplegia |
T
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| D. Hypopyrexia |
F
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| E. Contralateral cranial nerve palsies |
F
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| 18. Features of general paresis (GPI) may include: | |
| A. ‘Rod cells’ |
T
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| B. Cortical thickening |
F
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| C. Thinning of the dura |
F
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| D. Perivascular lymphocytes |
T
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| E. Spirochaetes found in the brain |
T
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| 19. Causes of mononeuritis multiplex include: | |
| A. Sarcoidosis |
T
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| B. Bronchial carcinoma |
T
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| C. Leprosy |
T
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| D. Polyarteritis nodosa |
T
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| E. Trauma |
F
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| 20. Features of pseudo-bulbar palsy include: | |
| A. Exaggerated jaw jerk |
T
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| B. Emotional lability |
T
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| C. Wasting of tongue |
F
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| D. Facial muscles’ fasciculation |
F
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| E. Dysphonia |
T
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| 21. Possible causes of sudden blindness include: | |
| A. Vitreous haemorrhage |
T
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| B. Hysterical dissociation |
F
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| C. Acute glaucoma |
T
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| D. Methanol |
T
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| E. Prolapsed intervertebral disc |
F
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| 22. Principal outputs of the basal ganglia go to: | |
| A. Cerebral cortex |
F
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| B. Red nucleus |
T
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| C. Tectum |
T
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| D. Subthalamic nucleus |
T
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| E. Substantia nigra |
T
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| 23. Functions of the limbic system may include: | |
| A. Emotional behaviour |
T
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| B. Motivation |
T
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| C. Sexual activity |
T
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| D. Conditioned reflexes |
T
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| E. Memory |
T
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| 24. Diencephalic structures include: | |
| A. Pons |
F
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| B. Pituitary |
T
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| C. Thalamus |
T
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| D. Cerebellum |
F
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| E. Hypothalamus |
T
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| 25. Functions of the reticular formation include: | |
| A. Arousal |
T
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| B. Principal input to basal ganglia |
F
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| C. Sleep |
T
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| D. Vigilance |
F
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| E. Principle input to limbic system |
T
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| 26. Features of the Brown-Sequard syndrome include: | |
| A. contralateral loss of conscious kinaesthesia |
F
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| B. Ipsilateral loss of crude touch |
F
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| C. Contralateral loss of two-point discrimination |
F
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| D. Ipsilateral loss of temperature |
F
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| E. Incomplete lateral hemisection of spinal cord |
F
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| 27. Branches of the basilar artery include: | |
| A. Posterior cerebral artery |
T
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| B. Anterior cerebral artery |
F
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| C. Posterior inferior cerebellar arteries |
F
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| D. Anterior inferior cerebellar arteries |
T
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| E. Labyrinthine arteries |
T
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| 28. Possible features of complete spinal cord transection include: | |
| A. Reflexes initially hyperactive |
F
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| B. Loss of all voluntary movement below lesion |
T
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| C. Development of automatic bladder in first 3 days usually |
F
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| D. Loss of all perception of sensation below lesion |
T
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| E. Loss of all reflexes after about three weeks |
F
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| 29. Possible components of the cerebellum include: | |
| A. Caudate nucleus |
F
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| B. Dentate nucleus |
T
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| C. Lentiform nucleus |
F
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| D. Vermis |
T
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| E. Cingulate gyrus |
F
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| 30. Features of anterior cerebral artery occlusion could include: | |
| A. Contralateral lower limb sensory deficits |
T
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| B. Contralateral hemianopia |
F
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| C. Clouding of consciousness |
T
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| D. Motor aphasia |
F
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| E. Contralateral lower limb paresis |
T
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| 31. Dopaminergic cell bodies are situated in: | |
| A. ventral tegmental area |
T
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| B. Substantia nigra |
T
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| C. Median raphe nucleus |
F
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| D. Dorsal raphe nucleus |
F
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| E. Arcuate nucleus of hypothalamus |
T
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| 32. Telencephalic structures include: | |
| A. Oculomotor nerves |
F
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| B. Basal ganglia |
T
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| C. Crura cerebri |
F
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| D. Optic nerves |
F
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| E. Cerebral hemispheres |
T
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| 33. Functions of astrocytes might include: | |
| A. Myelin sheath production |
F
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| B. Filling the role of fibrous tissue |
T
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| C. Lining the cerebral ventricles |
F
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| D. Most numerous of the glial cells |
T
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| E. Lining the spinal canal |
F
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| 34. Maxillary division branches of the trigeminal ganglion include: | |
| A. Supraorbital nerve |
F
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| B. Infraorbital nerve |
T
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| C. Superior alveolar nerve |
T
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| D. Inferior alveolar nerve |
F
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| E. Nasociliary nerve |
F
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| 35. Components of the basal ganglia may include: | |
| A. Amygdaloid nucleus |
T
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| B. Red nucleus |
F
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| C. Dentate nucleus |
F
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| D. Caudate nucleus |
T
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| E. Subthalamic nucleus |
F
|
| 36. Features of carotid sinus stimulation may include: | |
| A. Hyperventilation |
F
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| B. Hypertension |
F
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| C. Raised intracranial pressure |
F
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| D. Peripheral vasodilation |
T
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| E. Bradycardia |
T
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| 37. Components of the cerebellum include: | |
| A. Flocculonodular node |
T
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| B. Alveus |
F
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| C. Stria terminalis |
F
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| D. Fastigial nucleus |
T
|
| E. Interpositus nucleus |
T
|
| 38. Components of the Papez circuit include: | |
| A. Fornix |
T
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| B. Mamillary body |
T
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| C. Hippocampus |
T
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| D. Thalamus |
T
|
| E. Cingulate gyrus |
T
|
| 39. Possible components of the Pons include: | |
| A. Abducens nucleus |
T
|
| B. Oculomotor nucleus |
F
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| C. Trapezoid body |
T
|
| D. Red nucleus |
F
|
| E. Corpora quadrigemina |
F
|
| 40. Causes of pre-senile dementia include: | |
| A. Jakob-Creutzfeldt disease |
T
|
| B. Simple schizophrenia |
F
|
| C. Subacute spongiform encephalopathy |
T
|
| D. Manic-depressive psychosis |
F
|
| E. Punch-drunk syndrome |
T
|
| 41. Possible components of the pyramidal system include: | |
| A. Pyramidal tract |
T
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| B. Vestibular nuclei |
F
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| C. Anterior horn cells |
T
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| D. Cerebellum |
F
|
| E. Cortico-spinal tract |
T
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| 42. Components of the direct and consensual light reflexes could include: | |
| A. Superior colliculus |
F
|
| B. Lateral geniculate body |
F
|
| C. Ciliary ganglion |
T
|
| D. Visual cortex |
F
|
| E. Constrictor muscles of iris |
T
|
| 43. Components of the limbic system may include: | |
| A. Internal capsule |
F
|
| B. Median forebrain bundle |
T
|
| C. Corpus striatum |
F
|
| D. Isthmus |
T
|
| E. Medial lemniscus |
F
|
| 44. The circle of Willis is formed by: | |
| A. Superior cerebellar artery |
F
|
| B. Posterior spinal artery |
F
|
| C. Posterior communicating artery |
T
|
| D. Middle cerebral artery |
F
|
| E. Anterior inferior cerebellar artery |
F
|
| 45. Posterior cerebral artery occlusions cause: | |
| A. Contralateral hemianalgesia |
T
|
| B. Ipsilateral hemianaesthesia |
F
|
| C. Spontaneous pain |
T
|
| D. Ipsilateral hemiplegia |
F
|
| E. Ipsilateral hemianopia |
F
|
| 46. A left homonymous hemianopia may be due to a lesion in: | |
| A. Left optic tract |
F
|
| B. Right optic tract |
T
|
| C. Optic chiasma |
F
|
| D. Right lateral geniculate body |
T
|
| E. Left medial geniculate body |
F
|
| 47. Features of cerebellar disease include: | |
| A. Pendular nystagmus |
F
|
| B. Dysdiadochokinesis |
T
|
| C. Resting tremor |
F
|
| D. Past pointing |
T
|
| E. Scanning dysarthria |
T
|
| 48. Causes of papilloedema include: | |
| A. Central retinal vein thrombosis |
T
|
| B. Hypoparathyroidism |
T
|
| C. Cavernous sinus thrombosis |
T
|
| D. Hypercapnia |
T
|
| E. Cranial arteritis |
T
|
| 49. The following structures are in the pons: | |
| A. Reticular formation |
T
|
| B. Substantia nigra |
F
|
| C. Locus coeruleus |
T
|
| D. Trigeminal nerve nucleus |
T
|
| E. Vestibular nuclei |
T
|
| 50. The following structures are in the midbrain: | |
| A. Superior colliculus |
T
|
| B. Inferior colliculus |
T
|
| C. Substantia nigra |
T
|
| D. Ventral tegmental area |
T
|
| E. Medical longitudinal fasciculus |
F
|